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b,c, KL1 -mediated sensitization was observed in clinical isolates with high ( b ) and low ( c ) persister phenotypes (see Fig

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These findings support the relevance of MGBA mechanisms in BD pathophysiology, implicating both immune and metabolic pathways as shown in Table 8, and are conceptually illustrated through the microbiotaimmunemetabolicbrain axis model (Figure 6

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Most cancers exhibit a strong affinity for glutamine, and several cancer cell lines, such as glioblastoma, heavily rely on this amino acid for their survival, as they are unable to sustain themselves without an external source of supply [26]

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